Activation-repolarization mapping to guide VT-ablation without the need to induce the arrhythmia
Ventricular tachycardia and fibrillation are a major cause of sudden cardiac death, especially in the setting of chronic myocardial infarction (MI) . These arrhythmias are mostly based on re-entry  and depend on the combination of a trigger (usually a critically timed premature activation) and an arrhythmogenic substrate. Surviving myocardial bundles that penetrate the scar tissue form the substrate and are the basis for a long activation delays across the myocardial scar.  The site(s) where the wavefronts exit from the scar can be targeted by ablation therapy.